Als mensen tijd hebben om een emmer met ijskoud water over hun hoofd te gooien voor ALS, neem dan ook de tijd om dit artikel te lezen over ALS. Wat is ALS? Waarom blijken zoveel ALS patiënten Lyme te hebben? Het zou zeer nuttig zijn als we deze informatie gaan verspreiden: WHEN ALS IS LYME Examining the link between ALS and Neuroborreliosis Countries where Lyme is very rare also have very low ALS incidence Most ALS patients have classical symptoms of Neuroborreliosis Every few minutes, someone in the world dies of Amyotrophic Lateral Sclerosis. This book shows that most things we are told about ALS are incorrect: • ALS is not just a motor neurone disease but affects the entire brain • ALS is much more common in climates with endemic Ixodes ricinus ticks • The more Lyme disease reported in a US state, the more ALS cases too • The USs largest ALS clusterlies in the USs number one Lyme hotspot • Other majo ALS clusters are found in other major Lyme hotspots • Almost all ALS patients test positive for Lyme,when tested properly • Many ALS patients have the same reaction to antibiotics as a Lyme patient • There are dozens of documented ALS cases that cured using antibiotics • Antibiotictrials for ALS could never have succeeded -by design • Proper Lyme diagnosis to rule out ALS is sabotaged for financial reasons ALS is not a muscle disease Many people are unaware of the fact that ALS always only affects the central nervous system (brain and spinal cord), never the muscles directly. In Germany, ALS is generally called a Muskelkrankheit, a muscle disease. In the Netherlands, ALS is called the same, a spierziekte, a muscle disease. In Norway, ALS is called a muskelsykdom, again a muscle disease. However, ALS has nothing whatsoever to do with muscles. ALS is defined as a disease of the nerve cells in the brain and spinal cord (that control voluntary muscle movement). As we will explain, there are two fundamental things provably wrong with this definition, but the basic premisse is correct: Neurons are damaged. In up to 15% of cases, ALS patients suffer brain damage to the non-motor neuron parts of their brains so severe that it leads to a dual diagnosis such as ALS-dementia and in an additional 50% of ALS patients, there is still so much brain damage to the rest of the brain that it can be detected in a brain MRI. Research shows that at least two third of ALS patients suffer generalized brain damage in addition to motor neuron damage. (70% v dementie bleek Lyme, zie onderzoeken v oa Alan MacDonald en de plakken in de hersenen met DNA Borrelia Lyme) In case of sporadic ALS, the underlying cause is unknown, making ALS not a disease but a symptom description. It is crucial to keep this in mind. It is unfair to patients when they are lead to believe that they are with 100% likelihood suffering from a disease without a cure, instead of a perhaps treatable symptom of an identifiable illness. One of the reasons why people still believe that ALS is a disease is that ALS is also known as “Lou Gehrig’s disease”.When ALS got that name, it was not yet clear that ALS can be caused by a variety of factors, and that it is assumed to be a symptom of an unknown disease or -diseases. Even though there are countless cases of ALS being caused by Lyme disease, the medical establishment still believes that there is such a thing as “real ALS” and that it has an unknown cause or causes. So far, the only identified causes of Amyotrophic Lateral Sclerosis (Motor neuron damage) have been familial ALS and Lyme neuroborreliosis. Patients should understand that ALS is a symptom, and that ALS has been cured many times with antibiotic treatment. Our book documents eleven ALS patients who cured their ALS with antibiotics. These people have been diagnosed by a neurologist as having ALS, but it turned out their ALS was a symptom of Lyme disease and antibiotics provided a cure. When we compare these figures with the CDCs Lyme-risk map, we see that Minnesota has a higher Lyme risk than Washington, and Washington a higher Lyme risk than Texas. This corresponds with an increasing ALS risk for those states. Coincidence? A Minnesotan is twice as likely as a Texan to get ALS. And that same Minnesotan is as least twice as likely to contract Lyme disease. The more Ixodes ticks, the more ALS. (er zijn over de 300 verschillende stammen vd Borrelia Lyme) We see the lowest ALS-incidence in countries with few Ixodes ticks, and an ALS-incidence of up to three times that in areas with a high prevalence of Ixodes ricinus. A study by Sabelet al found Finnish ALS clusters related to place-of-birth and place-of-death.Those clusters were situated in the southeast of Finland, the area with the highest prevalence of ticks and Lyme disease. Cluster 1 in the rightmost picture (place of death) had double the ALS incidence as statistically expected1 The largest ALS cluster to have been identified so far is amongst employees of Kelly Air Force Base, decommissioned in 2001. The families of the victims declared to investigators that a total of 140 employees died of ALS, but because the death certificates usually mentioned aspiration pneumonia or similar ALS-related causes of death, the health authorities refused to recognize those deaths as ALS, and the figure was therefore spun down to 39 deaths - still highly anomalous ALS does not just plague Italian soccer players: Playing soccer is just as risky for Brits. (teken zitten ook vaak op voetbalvelden en veldjes) Countries where Lyme is very rare also have very low ALS incidence Mexico has a 37 times lower ALS incidence than Finland23, and Mexico also has an extremely much lower Lyme incidence as Finland.24 He added that typically, if people have Lyme disease for longer than the four week average associated with the disease, theyre instead treated for other diseases such as fibromyalgia or chronic fatigue syndrome. ALS clusters prove an environmental, not genetic cause A French study found nine conjugal cases of ALS, of which at least eight cases were sporadic ALS. Statistically speaking, this was highly unlikely if ALS is directly or indirectly caused by genetical factors, because the partners were not related to eachother. What remains as a plausible cause is either a local environmental toxin or an infectious agent. The geographical spread is relatively wide. It seems more likely that an infectious disease such as Lyme disease is responsible than some mysterious toxin that only affects a few couples here & there. Lyme disease is highly endemic in France and the couples could have been exposed to ticks or other vectors in their immediate surroundings. From the study Most ALS patients have classical symptoms of Neuroborreliosis It has always been a persistent myth that ALS is a motor neuron disease. This has recently been shown not to be true. In reality, most people with ALS suffer brain damage throughout the brain - its only that the damage to motor neurons is much more quantifyable and life-threatening than damage to the rest of the brain. In up to 15% of cases, the miscellaneous brain damage is so severe that it leads to a dual diagnosis such as ALS-dementia, but in an additional 50% of ALS patients, there is still so much brain damage to the rest of the brain that it can be detected in a brain MRI33 34. Research shows that at least two third of ALS patients suffer generalized brain damage in addition to motor neuron damage.35 Generalized neurological damage to cognition and behavior is typical for Lyme neuroborreliosis - and this is exactly the damage done to at least two thirds of ALS patients - its just that their paralysis overshadows it. Sanjay Kalra, a researcher in the facultys Division of Neurology and a practising neurologist, has published two papers in 2011 in the American Journal of Neuroradiology, providing evidence that ALS affects more than just the motor cortex, the part of the brain responsible for motor function. ALS was previously thought to be a disease restricted to the motor system causing only weakness, says Kalra, the principal investigator in both peer-reviewed papers. But a significant proportion of people with ALS also have cognitive and behavioural changes. If ALS is a manifestation of Lyme neuroborreliosis, one would indeed expect CNS damage in ALS patients not to be limited to motor neurons. Hard evidence links Lyme to ALS On ALS forums, it is almost common knowledge that Everyone tests positive for Lyme when the tests are sensitive enough, such as with the IGeneX test, that also includes bands 31 and 34, which are most specific for Borrelia and are excluded by other Lyme tests manufacturers. The below two ALS patients on als.net had positive Lyme tests In 2009, a Florida lab found mosquitoes carrying the Lyme spirochete. Fleas are carriers as well. Lida Mattmann found Borrelia spirochetes in tears. Borrelia has been found in perspiration, the placenta, blood and semen. Borrelia bacteria have even been found in dust from Africa 21 out of 24 ALS patients that tested Lyme-positive, making it 88%, or almost nine out of ten patients. Since the false-negative rate of the tests used is notoriously high, we are justified in concluding that most likely, every single ALS patient in their study was Lyme-positive. According to the Halperin study, American ALS patients have about a thousand times greater chance to be Lyme-positive than the average American. Not 1000 percent, mind you. 1000 times. That is a 100000% greater occurence of being Lyme-positive. A hundred thousand percent more. And even though their conclusion is that Lyme has nothing to do with ALS because they consider their findings to be coincidental, we think their findings speak for themselves. Nine out of ten ALS patients were infected with the Lyme spirochete, whereas only one in a hundred random people are. Coincidence? Around 25% of ALS starts as bulbar ALS, and those patients often deteriorate faster and often die from aspiration pneumonia. Bulbar means that the sclerosis happens in the lower motor neurons in the brain stem. Damage there causes problems swallowing, speaking and chewing. At least 12% of bulbar ALS patients have Borrelia bacteria in their brain The article talks about a patient with both upper and lower motor neuron disease. He tested positive for Lyme disease, in spite of not recalling a tick bite.Two weeks of Doxycycline and five days of Cefotaxime did not help him, but after having been given two weeks of Ceftriaxone he steadily improved and his diagnosis was changed from ALS to Lyme. But when they sought a second opinion from an infectious disease specialist knowledgeable about Lyme, they obtained the correct diagnosis of Lyme neuroborreliosis, received antibiotic treatment and their ALS disappeared. His doctors however managed to detect Borrelia DNA fragments in the patients urine This illustrates the importance of doing an antibiotic-provocative urine PCR in order to avoid false-negatives In case of ALS, it is essential to find a real specialist - someone aware of the proven medical fact that brain infections can cause ALS. People who cured their Lyme-caused ALS by taking antibiotics (lees artikel) Make sure proper Lyme tests are done - they never are! They do what the negotiated contracts with the insurance companies tells them to. They do what their experts tell them to. They prescribe Big Pharmas expensive, patented symptom relievers. The doctor who experiments can look forward to a malpractice lawsuit. The medical industrial complex is run by multinational pharmaceutical corporations and only those medical professionals that follow the party line have a chance of a career Effective treatment of late stage ALS is possible with aggressive antibiotic therapy that must include metronidazole. (ALS patients should not be treated with simple textbook antibiotic therapy which does not include a nitroimidazole.) Read more, Lees alle informatie, bewijzen en onderzoeken en verhalen van ALS patiënten zelf op: als-cure/ALS.pdf
Posted on: Thu, 18 Dec 2014 14:16:06 +0000
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