Irrespective of whether we are confronting a vaccine-derived hybrid strain of bacterial Meningitis here, or a laboratory-derived hybrid strain of the Ebola Virus (essentially a composite of the laboratory produced Aids Virus) the so-called vanguard of Health “Authority”, chiefly the CDC & WHO, are purposefully focusing on a Vaccine & Drug response to this current health crisis, while ignoring the fact that the Ebola virus can only thrive in a body starved of Selenium; that in fact ALL Ebola Virus victims, those who typically succumb to Hemorrhagic fever, viral replication (infected blood) and excessive hemorrhaging of the blood leading to death, concurrently suffer from acute malnutrition, marked by extreme Selenium deficiency in the body – thereby depriving our communities of critical information in a time of national emergency. ‘Ebola is classified as a “hemorrhagic fever” virus, and produces the characteristic hemorrhaging due to the formation of blood clots (“disseminated intravascular coagulation”), leading to the obstruction and rupture of small blood capillaries. It is very well documented that Se (Selenium) plays a significant role in the regulation of blood clotting via its effects on the thromboxane/prostacyclin ratio. Se (Selenium) has an anti-clotting effect, whereas Se (Selenium) deficiency has a pro-clotting or thrombotic effect. Se (Selenium) deficiency has been associated with thrombosis and even hemorrhaging, which has been documented in a number of animals with severe Se (Selenium) deficiency (often artificially induced), but is almost never seen in humans, probably because such an extreme Se (Selenium) deficiency is rarely attained due to the diversity of human diet. Our analysis suggests that severe Ebola infections could produce an artificial and extreme Se (Selenium) depletion, resulting in extensive cellular damage due to lipid peroxidation, combined with enhanced thrombosis. This could also contribute to the associated immune deficiency that has been observed in Ebola infections.‘ Note: ‘ If viruses like HIV-1 (Aids), coxsackievirus B3 (linked to Meningitis) and Ebola do encode selenoproteins, why does all the evidence suggest that dietary Se (Selenium) inhibits viral replication, whereas Se (Selenium) deficiency triggers replication?‘ Journal of Orthomolecular Medicine, 1995 Excerpt from upcoming VRM: The Ebola Report vaccineresistancemovement.org/?page_id=11240
Posted on: Wed, 08 Oct 2014 01:20:44 +0000
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