LARD PROMOTION IS A BIG MISTAKE OF WAPF: THE PROBLEM IS NOT ONLY HIGH CONTENT OF N-6 PUFA, BUT ALSO VERY HIGH CONTENT OF MUFA Omega-6 Fats: Excerpt from the Paul Jaminet´s article THE TROUBLE WITH PORK, PART 2: Omega-6 fats are a health villain: Excess omega-6 contributes to general inflammation, fatty liver disease, metabolic syndrome, obesity, and impaired immune function. And pork can be a major source of omega-6 fats. Nutritiondata lists the omega-6 fraction of lard at 11%. But the omega-6 fraction can be highly variable, depending on the pig’s diet. Chris Masterjohn recently reported that the lard used in the “high-fat” research diet was 32% polyunsaturated, nearly all of it omega-6: The graph shows the difference between the actual fatty acid profile as determined by direct analysis of the lard and the previously reported fatty acid profile, which had been estimated using the USDA database. We can see that the actual fatty acid profile is much higher in PUFAs, at the expense of both saturated and monounsaturated fats. In fact, the company had originally estimated the diet to provide 17 percent of its fat as PUFA, but now estimates it to provide a whopping 32 percent! ........................................................... Very good article but I could add a few points. Lard contains 56-62 % of unsaturated fats composed mainly from of OLEIC ACID (44-47%) and LINOLEIC ACID (6-10%) . (en.wikipedia.org/wiki/Lard). As pointed out by Jamines, Chris Masterjohn recently reported that the lard used in the “high-fat” research diet was 32% polyunsaturated, nearly all of it omega-6. Both monounsaturated (MUFA) and polyunsaturated (PUFA) fatty acids are the most unstable and prone to oil rancidity. Rancid oils are a major source of destructive free radicals in the human diet. Oils oxidize through exposure to air, heat and light and become unstable and highly toxic to your body. It is well established, that oleic acid (OA) induces rat VASCULAR SMOOTH MUSCLE CELL (VSMC) proliferation, migration and plays a central role in obesity and fatty acid-induced atherosclerosis. Recent study showed that OLEIC ACID BUT NOT PALMITIC ACID INDUCES PROLIFERATION OF HUMAN VASCULAR SMOOTH MUSCLE CELL PROLIFERATION AND MIGRATION (onlinelibrary.wiley/doi/10.1111/j.1582-4934.2010.01099.x/full) Another study showed that OA induces smooth muscle FOAM CELL formation and enhances atherosclerotic lesion development via CD36. Moreover, the OA-induced acceleration of atherosclerosis was reversed by reducing plasma FFA levels using the antilipolytic agent acipimox. An accumulation of foam cells in the arterial wall is the first visible sign of atherosclerosis and is termed fatty streak. According to the so-called oxidation hypothesis, one of the initial steps in atherogenesis is the oxidative modification of LDL and the uptake of the modified lipoprotein particles by macrophages, which in turn become lipid laden cholesterol-rich cells, so-called foam cells. (ncbi.nlm.nih.gov/pmc/articles/PMC3083368/)
Posted on: Wed, 17 Jul 2013 16:56:37 +0000
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