The Ebola & Aids (HIV) Virus share one precise common thread, a key protein capable of shutting down the body’s Immune system, by disabling what are known as T-cells. ‘T cells contribute to immune defenses in two major ways: Some direct and regulate immune responses…by communicating with other cells. Some stimulate nearby B cells to produce antibodies, others call in microbe-gobbling cells called phagocytes, and still others activate other T cells.‘ National Institute of Allergies & Infectious Diseases “You know the (Ebola) virus, it’s like ‘shock and awe.’ It’s like over within a week. I mean the virus grows very quickly and it kills off the very cells you need to mount your immune response.” Dr. Kartik Chandran, Whitehead Institute, Harvard Medical School, Albert Einstein College of Medicine ‘Ebola virus’s ability to enter cells is reminiscent of the Trojan Horse used by the ancient Greeks to defeat their archenemies. Ebola virus binds to the host cell’s outer membrane, and a portion of host cell membrane then surrounds the virus and pinches off, creating an endosome — a membrane-bound bubble inside the cell (see image). Endosomes carry their viral stowaways deep within the cell and eventually mature into lysosomes — tiny enzyme-filled structures that digest and recycle cellular debris. The viruses captive in the lysosome manage to escape destruction by exploiting components of the cell to gain entry to the cytoplasm, the substance between the cell membrane and the nucleus where the virus can replicate. But the identities of many of these components have remained unknown.‘ Albert Einstein College of Medicine, 08/24/2011 ‘Our screen uncovered 67 mutations disrupting all six members of the homotypic fusion and vacuole protein-sorting (HOPS) multisubunit tethering complex, which is involved in the fusion of endosomes to lysosomes, and 39 independent mutations that disrupt the endo/lysosomal cholesterol transporter protein Niemann–Pick C1 (NPC1).‘ Ebola virus entry requires the cholesterol transporter Niemann–Pick C1, International Weekly Journal of Science, 08/24/2011 ‘Niemann-Pick disease type C1 is a lysosomal storage disease caused by a mutation in the NPC1 gene. NPC1 encodes an integral membrane protein containing sequence motifs thought to be consistent with a role in intracellular transport of cholesterol to post-lysosomal destinations. Mutations in NPC1 have also been linked with obesity, HIV-AIDS, and Ebola virus.‘ The current virulent strain of the Ebola Virus is a (weaponized) variant of Yellow Fever Virus, invariably crossed with Malaria, Bacterial Septicemia (Septic shock) & HIV Aids. We have come to the end of the road; the culmination of decades of the most toxic vaccines ever created, gestating and mutating throughout the population of Sub-Saharan Africa – manifesting in the coveted Virus-Bacterial hybrid the Rockefellers have sought for decades. There is no plausible explanation for the presence of a T-Cell disrupting protein epitope (identical to the Aids Syndrome signature HIV component – designed specifically to target the Human Immune System) embedded in the current Ebola Virus strain: ‘Mutations in NPC1 (Niemann–Pick C1) have also been linked with HIV-AIDS, and Ebola virus.‘ Excerpt from VRM: Ebola Report vaccineresistancemovement.org/?p=13982
Posted on: Wed, 05 Nov 2014 15:14:56 +0000
Trending Topics
Recently Viewed Topics
© 2015