A high-carbohydrate diet creates excess insulin (no matter whether - TopicsExpress



          

A high-carbohydrate diet creates excess insulin (no matter whether these are simple or complex carbohydrates) and therefore activates CDDS with all its documented pathological degenerative effects. This is explicitly why a 30–50% carbohydrate intake is part of an optimal long-term Phase 1.5 dietary strategy to prevent this accelerated aging process. High fructose or galactose also increases insulin levels both indirectly and directly, especially with high-fructose corn syrup. Recently, high-fructose corn syrup has been labeled as a main dietary culprit in activating insulin resistance, and ultimately CDDS, since the 1980s when it was massively introduced into the diet of the general population. The healthiest carbohydrates, as I mentioned previously, are the nonstarchy, fibrous carbohydrates in vegetables, leafy greens, sprouts, and sea vegetables. Insulin resistance, which indirectly raises insulin in the blood as a result, prevents a burning of fats for energy because the role of insulin is to store fat as energy. When I talk about the idea of cutting down on carbohydrates, it helps to understand that, physiologically, there is no absolute essential need for carbohydrates, but a 25–45% carbohydrate intake of nonstarchy vegetables, greens, sea vegetables, and sprouts gives an optimal healthy carbohydrate input, therefore minimizing insulin secretion and therefore decreasing insulin resistance. It also helps us shift to an emphasis on fat metabolism instead of carbohydrate metabolism as our fundamental energy source. Because all carbohydrates increase insulin secretion, a high carbohydrate intake essentially accelerates insulin resistance, CDDS, and consequently the rate of aging. Gabriel Cousens, MD There Is a Cure for Diabetes (Revised Edition) Could you or someone you love benefit from Dr. Cousens Diabetes Recovery Program? July 17th is our next program and space is limited. Visit ow.ly/xp9ed for more information!
Posted on: Wed, 11 Jun 2014 02:00:17 +0000

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