take c24/7 everyday The threat of Free Radicals Cells are - TopicsExpress



          

take c24/7 everyday The threat of Free Radicals Cells are continually under attack from compounds called free radicals. Generated as a by-product of the bodys own metabolism or as a result of external factors like pollution, free radicals can kill cells by damaging the membrane, the enzymes and the DNA contained within. Free radicals pose a particular risk to the cells of the nervous and immune systems, and they are even thought to contribute to the progression of many diseases and the onset of premature ageing. The threat of Oxidative stress Oxidative stress is an attack on cells when a biological system cannot readily detoxify the redox reactive intermediates or easily repair the resulting damage. Oxidative stress is involved in many diseases, such as atherosclerosis, Parkinsons disease, heart failure, myocardial infarction, Alzheimers disease, fragile X syndrome and chronic fatigue syndrome, but short-term oxidative stress may also be important in prevention of aging by induction of a process named mitohormesis. How antioxidants help? Antioxidants help prevent the free radicals from damaging cells. Whilst the bodys normal antioxidant defences provide some protection, the addition of antioxidants to the diet provides an additional active shield that supports those natural defences. Antioxidants are free radical scavengers: they slow or prevent the oxidation of other molecules by capturing free radicals. Oxidation is a chemical reaction that transfers electrons from a substance to an oxidizing agent. Oxidation reactions can produce free radicals, which start chain reactions that damage cells. Antioxidants terminate these chain reactions by removing free radical intermediates, and inhibit other oxidation reactions by being oxidized themselves. As a result, antioxidants are often reducing agents such as thiols or polyphenols. Fight oxidative stress and free radicals! Take C24/7 Natura- ceuticals. pm me for order
Posted on: Wed, 10 Sep 2014 23:43:50 +0000

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